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2015). These research, taken jointly, recommend that, Though a direct activation of GPR55 or PPAR?α takes place, PEA can develop its anti?inflammatory action in the gut also via indirect activation of CB1 and CB2 receptors, most likely due to the ability of this compound to potentiate the

2016a). This more compact enhance was proportionate to the scaled-down peak of PEA concentrations detected in human volunteers in contrast with beagle pet dogs. This big difference, in turn, may be brought on by the lessen overall level of PEA acutely administered to human volunteers (about &sim

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